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phospho parkin ser65  (Bioss)


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    Structured Review

    Bioss phospho parkin ser65
    Phospho Parkin Ser65, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 11 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/parkin+(ser65)+polyclonal+antibody/Parkin+(Ser65)+Polyclonal+Antibody/pm41654147-170-66-69
    Average 94 stars, based on 11 article reviews
    phospho parkin ser65 - by Bioz Stars, 2026-09
    94/100 stars

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    Related Articles

    Incubation:

    Article Title: Nrf2 alleviates excessive deposition of extracellular matrix in mammary fibrosis through TGF/Smad and ROS signals
    Article Snippet: .. After transfer, membranes were blocked and incubated overnight at 4 °C with primary antibodies against Hsp60 (15282-1-AP; Proteintech, China), Mfn2 (12186-1-AP; Proteintech, China), P62 (18,420-1-AP; Proteintech, China), PHB2 (12295-1-AP; Proteintech, China), TGF-β1 (21898-1-AP; Proteintech, China), Parkin (14060-1-AP; Proteintech, China), p-Parkin (bs-19882R; Bioss, China), LC3 (14600-1-AP; Proteintech, China), Smad (AF6367; Affinity, China), p-Smad (AF3367; Affinity, China), Vimentin (AF7013; Affinity, China), α-SMA (AF1032; Affinity, China), E-cadherin (A3044; Abclonal, China), Collagen1 (A16891; Abclonal, China), Pink1 (BC100-494; Novus Biologicals, USA), and β-actin (66009-1-Ig; Proteintech, China). .. Membranes were then incubated with horseradish peroxidase (HRP)-conjugated secondary antibodies (1:5000; Santa Cruz, CA, USA) for 1 h at room temperature.

    Article Title: Bisphenol A Disrupts Spermatogenesis via Excessive Mitophagy-Driven Ferroptosis.
    Article Snippet: BPA is a globally ubiquitous industrial compound that harms male reproductive health by causing abnormal sperm development and subsequent spermatogonia loss, yet the underlying mechanisms of BPA-induced spermatogenesis disorder remain unclear.. Here, we explored BPA’s effects on adolescent male mice and GC-1 cells by gavaging mice with BPA at doses of 20, 200, or 2000 μg/kg/d for 4 weeks and treating GC-1 cells with 10 μM BPA for 12 h to establish a damage model.. The results revealed that BPA induced spermatogenesis disorder via ferroptosis, which was associated with the activation of excessive mitophagy.

    Article Title: Nrf2 alleviates excessive deposition of extracellular matrix in mammary fibrosis through TGF/Smad and ROS signals.
    Article Snippet: .. After transfer, membranes were blocked and incubated overnight at 4 °C with primary antibodies against Hsp60 (15282-1-AP; Proteintech, China), Mfn2 (12186-1-AP; Proteintech, China), P62 (18,420-1-AP; Proteintech, China), PHB2 (12295-1-AP; Proteintech, China), TGF-β1 (21898-1-AP; Proteintech, China), Parkin (14060-1-AP; Proteintech, China), p-Parkin (bs19882R; Bioss, China), LC3 (14600-1-AP; Proteintech, China), Smad (AF6367; Affinity, China), p-Smad (AF3367; Affinity, China), Vimentin (AF7013; Affinity, China), α-SMA (AF1032; Affinity, China), E-cadherin (A3044; Abclonal, China), Collagen1 (A16891; Abclonal, China), Pink1 (BC100-494; Novus Biologicals, USA), and β-actin (66009-1-Ig; Proteintech, China). .. Membranes were then incubated with horseradish peroxidase (HRP)-conjugated secondary antibodies (1:5000; Santa Cruz, CA, USA) for 1 h at room temperature.

    Article Title: Carbon disulfide induces accumulation of TDP-43 in the cytoplasm and mitochondrial dysfunction in rat spinal cords.
    Article Snippet: The relationship between environmental neurotoxicant exposure and neurodegenerative diseases is being extensively investigated.. Carbon disulfide, a classic neurotoxicant and prototype of dithiocarbamates fungicides and anti-inflammatory agents, has been detected in urban adults, raising questions about whether exposure to carbon disulfide is associated with a high incidence of neurodegenerative diseases.. Here, using rat models and SH-SY5Y cells, we investigated the possible mechanistic linkages between carbon disulfide neurotoxicity and the expression of TDP-43 protein, a marker of amyotrophic lateral sclerosis/frontotemporal lobar degeneration.

    Membrane:

    Article Title: Bisphenol A Disrupts Spermatogenesis via Excessive Mitophagy-Driven Ferroptosis.
    Article Snippet: BPA is a globally ubiquitous industrial compound that harms male reproductive health by causing abnormal sperm development and subsequent spermatogonia loss, yet the underlying mechanisms of BPA-induced spermatogenesis disorder remain unclear.. Here, we explored BPA’s effects on adolescent male mice and GC-1 cells by gavaging mice with BPA at doses of 20, 200, or 2000 μg/kg/d for 4 weeks and treating GC-1 cells with 10 μM BPA for 12 h to establish a damage model.. The results revealed that BPA induced spermatogenesis disorder via ferroptosis, which was associated with the activation of excessive mitophagy.

    Blocking Assay:

    Article Title: Bisphenol A Disrupts Spermatogenesis via Excessive Mitophagy-Driven Ferroptosis.
    Article Snippet: BPA is a globally ubiquitous industrial compound that harms male reproductive health by causing abnormal sperm development and subsequent spermatogonia loss, yet the underlying mechanisms of BPA-induced spermatogenesis disorder remain unclear.. Here, we explored BPA’s effects on adolescent male mice and GC-1 cells by gavaging mice with BPA at doses of 20, 200, or 2000 μg/kg/d for 4 weeks and treating GC-1 cells with 10 μM BPA for 12 h to establish a damage model.. The results revealed that BPA induced spermatogenesis disorder via ferroptosis, which was associated with the activation of excessive mitophagy.

    other:

    Article Title: Staphylococcus aureus induces mitophagy to promote its survival within bovine mammary epithelial cells.
    Article Snippet: Mitophagy occurs in a variety of pathogenic infections.. However, the role of mitophagy in the intracellular survival of Staphylococcus aureus (S.aureus) within bovine mammary epithelial cells (BMECs) and which molecules specifically mediate the induction of mitophagy remains unclear.. Therefore, this study aims to investigate the role and mechanism of mitophagy in the intracellular survival of S.aureus.

    Aqueous Normal-phase Chromatography:

    Article Title: MOTS-c attenuates cardiac dysfunction following high altitude exposure by promoting mitophagy.
    Article Snippet: This is a PDF of an article that has undergone enhancements after acceptance, such as the addition of a cover page and metadata, and formatting for readability.. This version will undergo additional copyediting, typesetting and review before it is published in its final form.. As such, this version is no longer the Accepted Manuscript, but it is not yet the definitive Version of Record; we are providing this early version to give early visibility of the article.

    Control:

    Article Title: MOTS-c attenuates cardiac dysfunction following high altitude exposure by promoting mitophagy.
    Article Snippet: This is a PDF of an article that has undergone enhancements after acceptance, such as the addition of a cover page and metadata, and formatting for readability.. This version will undergo additional copyediting, typesetting and review before it is published in its final form.. As such, this version is no longer the Accepted Manuscript, but it is not yet the definitive Version of Record; we are providing this early version to give early visibility of the article.



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